Multimodal electrophysiological analyses reveal that reduced synaptic excitatory neurotransmission underlies seizures in a model of NMDAR antibody-mediated encephalitis

Sukhvir K Wright, Richard E Rosch, Max A Wilson, Manoj A Upadhya, Divya R Dhangar, Charlie Clarke-Bland, Tamara T Wahid, Sumanta Barman, Norbert Goebels, Jakob Kreye, Harald Prüss, Leslie Jacobson, Danielle S Bassett, Angela Vincent, Stuart D Greenhill, Gavin L Woodhall

Aston Medical Research Institute Aston Medical School Aston University Birmingham UK
King's College London
Department of Paediatric Neurology
Great Ormond Street Hospital for Children NHS Foundation Trust
Science for Life Laboratory, KTH - Royal Institute of Technology, Stockholm SE-17121, Sweden; Department of Bioengineering, Marmara University, Istanbul, Turkey; Department of Bioengineering, Istanbul Medeniyet University, Istanbul, Turkey.
Center for Systems Genomics, Pennsylvania State University, University Park, Pennsylvania, USA.
Division of Neurology, The Children's Hospital of Philadelphia, Philadelphia, Pennsylvania.
Department of Gynecology and Obstetrics, University Hospital Düsseldorf, Heinrich-Heine University Düsseldorf, Düsseldorf, Germany.
German Center for Neurodegenerative Diseases (DZNE) Berlin
Oxford University Hospitals NHS Foundation Trust

Research output: Contribution to journal › Article › peer-review

22 Citations (Scopus)

Multimodal electrophysiological analyses reveal that reduced synaptic excitatory neurotransmission underlies seizures in a model of NMDAR antibody-mediated encephalitis

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Neuroscience

Pharmacology, Toxicology and Pharmaceutical Science